The kinetic models based on Ozawa’s and Mo’s methods were used to analyze the nonisothermal crystallization behaviors. It was found that the latter succeeded in describing the nonisothermal crystallization behavior of the PP containing UHMWPE, while the former was not appropriate. The activation energy for the nonisothermal crystallization determined by Kissinger’s method also indicated that the crystallization ability of PP was improved with the addition of UHMWPE. Owing to the modification of the crystallization kinetics of the PP materials by introduction of UHMWPE, the foaming properties
(i.e., cell uniformity and expandability AZ 628 etc.) were improved significantly. (C) 2010 Wiley Periodicals, Inc. J Appl Polym Sci 119: 1275-1286, 2011″
“Objective. BUBR1 is one of the key components of the spindle assembly checkpoint ( SAC) machinery and is activated in response to kinetochore tension. Defects in the SAC contribute to an increased rate of aneuploidization during tumorigenesis. The aim of the present study was to examine the immunohistochemical expression of BUBR1 protein for human oral squamous cell carcinogenesis.
Study design. A total of 120 samples of squamous cell carcinoma (SCC, n = 43) and
5 types of potentially malignant disorders (PMDs: oral epithelial dysplasia, n = PF00299804 11; hyperkeratosis/epithelial hyperplasia, n = 20; lichen planus, n = 16; submucous fibrosis, n = 19; and verrucous hyperplasia, n = 11) of human oral mucosa (1991-2001) from our institution were retrieved and immunohistochemical staining were performed. Normal oral mucosa ( n = 9) and fibrous hyperplasia ( n = 9) from patients without the aforementioned oral habits were also included in the study.
Results. BUBR1
staining was detected at the basal and suprabasal layers in 75 (97.4%) of 77 samples of PMD and 43 (100%) of 43 samples of SCC of oral mucosa but was absent in all samples of normal oral mucosa ( n = 9) and fibrous hyperplasia ( n = 9). BUBR1 expression of various types of PMD and SCC of oral mucosa was significantly overexpressed as compared respectively with normal mucosa ( P < .001) and fibrous hyperplasia ( P < .001). Moreover, the expression of oral SCC was significantly higher as compared respectively with the 5 types of oral PMD; on the other hand, BUBR1 expression of verrucous hyperplasia was significantly GSK461364 Cell Cycle inhibitor higher than that of the other 4 types of PMD of oral mucosa ( P < .001).
Conclusion. Our results may interpret that BUBR1 protein is suggested to be one of the contributing factors involved in the pathogenesis of oral SCC. These also hypothesize that BUBR1 protein is a putative biomarker for human oral squamous cell carcinogenesis. ( Oral Surg Oral Med Oral Pathol Oral Radiol Endod 2010; 109: 257-267)”
“We demonstrate that the electron injection barrier (Delta(e)) between Co and C-60 can be tuned by inserting a thin Alq(3) interlayer.