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25. Akyürek N, Memis L, Ekinci O, Köktürk N, Oztürk C: Survivin expression in pre-invasive lesions and non-small cell lung carcinoma. Virchows Arch 2006, 449: 164–170.CrossRefPubMed Competing interests The authors declare that they have no competing interests. Authors’ contributions Authors have made substantial contributions to conception and design (MC, MM, and SY), acquisition of data Anidulafungin (LY303366) (KT and KA), analysis, interpretation of data, organizing study (SY), and supervision of research group (KK)”
“Introduction As early as 1863, Virchow first

postulated that cancer originates at the sites of chronic inflammation. This is partly based on his hypothesis that some classes of irritants causing inflammation also enhance cell proliferation [1]. In the past decades, scientists have made considerable progress in research on the relationship between cancer and inflammation [1, 2]. Inflammation is a part of the host’s response to either internal or external environmental stimuli. This response serves to counteract the trauma incurred by these stimuli against the host. This can be pyrogenic, as indicated by fever. Acute inflammation or fever manifested for a short period has a therapeutic consequence [3]. Under normal circumstances, the wound healing process is considered an acute inflammation, like R406 price surgical wound healing. This process involves the classic model of inflammatory response, including the formation of granulation tissue, leukocyte infiltration, angiogenesis factor, and cytokines network [4]. During acute inflammation, the emergence of cell proliferation, angiogenesis, and reconstruction of the organization are very similar to tumor growth and progression.